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Both pathways are linked in some cell types by the cleavage of BID, a proapoptotic member of the Bcl-2 family of proteins, generating tBID in a process catalyzed by caspase-8 activated by the extrinsic pathways (Scaffidi et al., 1998)

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Therefore, we concluded that defective B cell development causes the decrease of MZB content in Gclc fl/fl Mb1-Cre + mice

Indian J Pharm Sci